Way back in May 2021 when the vaccines were ‘safe and effective’, friend Allan asked me how the shots may be causing heart attacks and other adverse events and what could be the long term effects of being mRNAed. Up to this time I was in a quandary as to who to believe: the health officials insistence of ‘totally safe’ and Allan’s paper research that said otherwise. Then a recently vaccinated friend died suddenly and unexpectedly of a massive heart attack. I started to dig deeper.
By the end of May 2021 it was publicly known how the mRNA was supposed to work. A snippet of mRNA with the genetic code needed to assemble spike proteins, those proteins the virus needs to attach to a cell’s surface so it will be taken into the cytoplasm. The idea was that the mRNAed cells would manufacture then display the spike proteins on their surface. With these foreign antigens so displayed, prowling T-lympocytes would recognize the proteins, label the cells as infected and release lymphokines to alert B-lymphocytes to start manufacturing antibodies: specific antibodies that would cling to and inactivate the spike proteins.
As with all vaccines, the logic was that if you were exposed to Covid, the already circulating vaccine induced antibodies would attach to the spike proteins of the coronavirus and prevent them from infecting cells.
From the start this didn’t seem to make a lot of sense. Why only one viral protein? Most effective vaccines use multiple proteins, even whole viral capsids or weakened or killed viruses. Single-stranded RNA is relatively unprotected and mutates readily: therefore evolution is rapid in RNA viruses. This is why there are few effective vaccines for RNA viruses and why earlier attempts to develop HIV and cold vaccines has been ineffective, and in some cases deadly. Using only one protein, and a spike protein that is highly mutable is a recipe for immune escape. This escape occurs as binding regions of the spike quickly evolve and selection pressure favours variants that evade the immune response.
With my limited understanding of viral evolution, I was able to predict that there would be new variants and the vaccine would quickly lose effectiveness.
Why also the spike protein? This is an active protein that has the potential to attach cell membranes together forming agglutinations of dysfunctional cells. Other less biologically active proteins may still have provoked an immune response without the potential side effects. Recent studies have shown that the fatigue of ‘long covid’ or ‘long vaccines’ may be due to the clumping of spike protein presenting red blood cells which prevents them from entering small capillaries. Less capillary perfusion lessens the pickup and delivery of oxygen to the tissues causing fatigue.
Also back in May 2021, there was circulating information about the vaccine leaking from the deltoid muscle injection site into the general circulation. It actually seems naive that health workers thought the mRNA would stay put and not travel through the vast capillary and lymphatic beds that surround all cells. The mRNA was carried in lipid nanoparticles (LPNs), fatty droplets that can merge with cell membranes and transfer the mRNA into the cell. Early studies showed that carried by the LPNs, the spike producing mRNA was able to travel through arterial highways to distant sites, including the liver, kidneys and ovaries.
When the T-lymphocytes identify infected cells, they also label these cells for destruction. The destroyers are the killer T-cells which are alerted, attach to the infected cells and secrete toxic enzymes into the cells to destroy their viral making machinery.
When the vaccine’s lipid nanoparticles enter the bloodstream is is probable that they will enter the thin layer of endothelial cells that line the arteries. These cells will display the spike proteins, the T-lymphocytes will identify the foreign proteins and label the cell for destruction. Along come the killer T-cells, they recognize the infected endothelial cells and destroy them.
Now a few destroyed cells is business as usual and we get on with living, but in a flood of billions of nanoparticles released into the bloodstream, billions of cells on the arterial surfaces may be infected and destroyed. Widespread damage to the arterial linings may occur with resulting inflammation as the arterial walls begin to repair themselves. High blood pressures within arteries tend to collapse blood capillaries within the arterial walls slowing repair and in some cases producing atheromas, fibrous, fatty scar tissue that grows into the opening of the artery.
With my limited understanding of cardiovascular physiology I was able in May 2021 to predict that there could be an long term increase in the number of heart attacks and strokes due to an increase in the number of atheromas caused by ‘vaccine’ related inflammation of the arterial linings. I predicted that the ‘canary in the coal mine’ would be high performance athletes who would collapse as their vaccine damaged circulatory systems shut down. A few weeks later, the first footballers suddenly died.
In the following interview by John Campbell, excellent evidence is given for the abnormal and now common presence of long fibrous clots in the arteries of the deceased.
Although there are competing theories of how the fibrous clots are formed, I am kind of pleased with my ability to predict. However, I am not writing this as a self serving “I told ya so!”, the whole thing leaves me mortified that it actually happened. After all, if a lapsed evolutionary zoologist/physiologist of a half century ago can make these predictions why were the current highfalutin biochemists, pharmacologists, geneticists, virologists, immunologists … unable to make the same predictions about this novel gene therapy? Predictions that should have been addressed before this biotravesty was foisted upon a global experimental cohort of billions.
Three possibilities: stupidity, incompetence or intent.
Scico 
Leave a comment